Sónia C. Correia
+ 351 239 820 190
sonia.correia@cnc.uc.pt
Postdoctoral researcher
Group at CNC
Education: 

In 2007, Sónia C. Correia obtained is degree in Biology from the Faculty of Sciences and Technology, University of Coimbra, Portugal. Then, Sónia C. Correia received her PhD in Cell Biology from University of Coimbra, Portugal, in 2012. Currently, she is a postdoctoral researcher at Center for Neuroscience and Cell Biology (CNC) with a fellowship from the Portuguese Foundation for Science and Technology. Sónia C. Correia’s research is mainly focused on deciphering the role of hypoxia up-regulated mitochondria movement regulator (HUMMR) on defective mitochondrial trafficking in Alzheimer’s disease and its potential as a therapeutic target to prevent or halt the progression of this devastating neurodegenerative disease. Despite her young age, Sónia C. Correia has already an impressive number of publications, as evidenced in her Curriculum Vitae that includes more than 40 scientific peer-reviewed and an h-index = 23  and > 1400 citations, according to Google Scholar. In 2010, Sónia C. Correia won the ASN Neuro Poster Prize sponsored by the American Society of Neurochemistry.

Afiliation: 

CNC

Area of Research: 

Unveiling the molecular mechanisms involved in Alzheimer's disease pathology and preconditioning-triggered brain tolerance, putting a special focus on the mechanisms mediated by mitochondria.

Research Summary: 
orcid.org/0000-0001-5360-2564
Selected Publications: 

Correia SC, Resende R, Moreira PI, Pereira CM. Alzheimer's disease-related misfolded proteins and dysfunctional organelles on autophagy menu! DNA Cell Biol. 2015;34:261-73. (IF: 2.055)

Correia SC, Carvalho C, Cardoso S, Santos RX, Plácido AI, Candeias E, Duarte AI, Moreira PI. Defective HIF Signaling Pathway And Brain Response To Hypoxia In Neurodegenerative Diseases: Not An "Iffy" Question! Curr Pharm Des. 2013;19:6809-22. (IF: 3.288)

Correia SC, Santos RX, Santos MS, Casadesus G, Lamanna JC, Perry G, Smith MA, Moreira PI. Mitochondrial abnormalities in a streptozotocin-induced rat model of sporadic Alzheimer's disease. Curr Alzheimer Res. 2013;10:406-19. (IF: 3.796)

Correia SC, Santos RX, Cardoso SM, Santos MS, Oliveira CR, Moreira PI. Cyanide preconditioning protects brain endothelial and NT2 neuron-like cells against glucotoxicity: role of mitochondrial reactive oxygen species and HIF-1α. Neurobiol Dis. 2012; 45:206-18. (IF: 5.624)

Correia SC, Santos RX, Perry G, Zhu X, Moreira PI, Smith MA. Insulin-Resistant Brain State: the culprit in sporadic Alzheimer's Disease? Ageing Res Rev. 2011; 10:264-73. (IF: 6.174)

Other information: 
 
   
© 2017 CNC - Center for Neuroscience and Cell Biology powered by ponto.C